A pulp polyp, also known as chronic hyperplastic pulpitis, is a “productive” (i.e., growing) inflammation of dental pulp in which the development of granulation. Pulp polyp (PP) is also known as chronic hyperplastic pulpitis or proliferative pulpitis. Clinically it appears as proliferative red mass seen in the occulsal portion . PDF | Chronic hyperplastic pulpitis (pulp polyps) usually occurs in molar teeth of children and young adults and is characterized by an.
|Published (Last):||10 October 2018|
|PDF File Size:||19.11 Mb|
|ePub File Size:||12.10 Mb|
|Price:||Free* [*Free Regsitration Required]|
Chronic hyperplastic pulpitis pulp polyps usually occurs in molar teeth of puloitis and young adults and is characterized by an overgrowth of granulomatous tissue into the carious cavity. Here, we report a rare type of pulp polyp in lower third molar of a year-old woman that not only grow into carious cavity but also extruded in very large size that interfered with occluding of the teeth.
Hyperplastic pulpitis is a type of irreversible chronic pulpitis that occurs usually in young teeth where the pulp has been exposed by caries or trauma 1. Mechanical irritation and bacterial invasion result in a level of chronic inflammation that produces hyperplastic granulation tissue extrudes from the chamber and often fills the associated dentinal defect 2.
Unusual Presentation of Chronic Hyperplastic Pulpitis: A Case Report
A year-old woman referred for treatment of left side lesion of the oral cavity. She gave history of six months for its presence that enlarged gradually and interfered with eating and occluding the hyperpllastic, so that made patient worried about it Figure 1Figure 2. Left side semi impacted carious lower 3 rd molar was the origin of the lesion. In review of systems ROS she gave history of idiopathic thrombocytopenic purpura and splenectomy of 12 years ago and caesarian section three years ago, but both of them had no relation to the present lesion.
Laboratory examinations including CBC differential, WBC, platelet count, PT, and PTT were within normal hyper;lastic, and in orthopanthogram radiography we observed left carious mesioangular semi impacted 3 rd molar with no specific lesion in bone and adjacent chronid Figure 1.
Intraoral examination showed a large polypoid lesion with about 3. So with considering the radiographic and clinical data these differential diagnoses were possible:. The surgical procedure for excisional biopsy of the lesion and surgical removal of 3 rd molar tooth performed under local anesthesia Persocaine-E, Darou Pakhsh-Tehran, Iran. We send the specimen included the carious 3 rd molar with base of the lesion in the carious cavity and the bulk of the lesion to the pathologist for histopathologic examination Figure 3.
Microscopic finding revealed a mass of inflamed granulation tissue resembling pyogenic granuloma that protruding from the. The fibrovascular stroma contained numerous capillary sized blood vessels lined by plump endothelial cells.
There were also significant mixed inflammatory cells composed of lymphocytes, plasma cells and neutrophils. Diagnosis was chronic hyperplastic pulpitis Figure 4Figure 5.
Hyperplastic pulpitis pulp polyp is the most visually dramatic of all pulp response, rising out of the carious shell of the crown and is a “mushroom” of living pulp tissue that is often firm and insensitive to touch 3. It is a response of the pulp to acute inflammation occurs in young teeth but never in teeth of old patients, may this be indicative of a good pulpal response. Presumably the young pulp does not become necrotic following exposure, because its natural defenses and rich supply of blood nyperplastic it to resist bacterial infection.
Transudates and exudates which are inflammatory response products in open chronic pulpitis, drain hyprrplastic the oral cavity and do not accumulate.
Thus intrapulpal pressure, which may consequently cause tissue damage and destruction of the microcirculation, does not develop 1. According to chronologic time for development of the lower 3rd molar teeth between 4. No severe pain because this type of lesions usually has no plupitis and subside within seconds after the stimulus is removed 2. No mobility of tooth and sensitivity to percussion because significant inflammation has not yet spread hcronic the apical area 2.
Fear of surgical procedures and assume a lesion as a cancer cancerophobia. In treatment planning for Oral and maxillofacial lesions, we must consider the clinical finding, and dental hyperplastkc of the patient and finally the histopathologic report for management of such lesions in spite of unusual size of them.
National Center for Biotechnology InformationU. Journal List Iran Endod J v. Author information Chroniic notes Copyright and License information Disclaimer. This article has been cited by other articles in PMC.
Abstract Chronic hyperplastic pulpitis pulp polyps usually occurs in molar teeth of children and young adults and is characterized by an overgrowth of granulomatous tissue into the carious cavity. Case Reports, Dental Pulp Diseases.
Open in a separate window.
The surface of the lesion was ulcerated and replaced by a fibrin purulent membrane. The large size of the lesion in this patient may be due to: Fear of surgical procedures and assume a lesion as a cancer cancerophobia 4. Neglected treatment by the patient for about six months 5. Good blood supply to the lesion 6. Continuous trauma from occluding the teeth.
Histological evaluation of teeth with hyperplastic pulpitis caused by trauma or caries: Oral and Maxillofacial Pathology. BC Decker Inc; Principle of management of impacted teeth. Principles of Oral and maxillofacial Surgery.
Peterson’s principle of Oral and Maxillofacial surgery. Support Center Support Center.